Antioxidant enzyme support and iron metabolism

Copper

Copper is an essential trace mineral required for superoxide dismutase function, iron metabolism, and connective tissue formation. Deficiency is uncommon but clinically significant; supplementation in replete individuals is not indicated.

Evidence Level B

Evidence and Risk Labels

Evidence A/B/C reflects research maturity, and risk levels reflect monitoring needs. These labels support comparison, not diagnosis or treatment decisions.

See full scoring guide

Clinical Snapshot

Effect Size
Deficiency correction: significant; routine supplementation in replete individuals: minimal to none
Safety
Moderate Risk

Excess copper is toxic; zinc supplementation >25 mg/day can deplete copper — monitor when zinc supplementing long-term. Wilson's disease is an absolute contraindication.

Research Dosing

This reflects common ranges and protocols used in published studies, not personal medical advice.

Typical Daily Dose
1-2 mg (as part of multimicronutrient or to counterbalance zinc)
Timing
With meals
Protocol Duration in Studies
Ongoing at low dose if zinc supplementing

Balance with zinc is critical: ratio of 8-15:1 (zinc:copper) is typically recommended.

Best Fit Profiles

  • deficiency correction; maintaining balance with zinc supplementation

Source Links